Read the paper. Salient part on conditions for fibrinolytic activity AFTER death:

"healthy state of the organism immediately before death seems to be an essential condition for development of fibrinolysis post-mortem. [...] fibrinolytic activity inblood after death to be the normal response to death of the healthy organism."

Further, as per the paper:

"presence in tissue of a fibrinolytic activator, fibrinokinase, and the activation of plasminogen by this activator has been described [...] blood from such cases was able to decompose fibrinogen and fibrin, has been amply confirmed in later works

The above is a normal process of blood fibrin formation under specific conditions.

However, the AMYLOID fibrin clots pulled out by the embalmers AND also pathologists are completely impervious to fibrinokinase and other fibrinolytic enzymes.

They are different. Both are fibrins, but the latter are amyloid structures consisting of high amount of β-rich amyloids, and especially such that they are distinct from the previously known amyloid rich fibrins to be labelled "anomalous".

Ref: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8883497/

Further point: If you have several embalmers and pathologists who have a combined experience of several decades and have NEVER seen a single issues of such anomalous amyloid fibrin clots (type, biochemical stability, length, location), but have seen hundreds of cases after the covid-19 injection started, I think the reasoning for correlation is quite strong.

Even the "mainstream" medical literature on PASC is confirming these anomalous amyloid fibrin clots (see above reference).

So not the same kind of clots and thus, most likely not the same etiology (compare with findings from 1st paper for findings on healthy subjects).