I also wanted to mention something else, I was going to make my own thread but meh, ill say it again, I am a terrible op. I just dont care about much lol.
I was googling the relationship between long covid and low phosphorous. The last time I looked into this, was about 1 and 1/2 years ago when I had omicron, and passed out and my blood pressure was hitting something like 100/50, heart rate 32 while the emts had me hooked up. It was brutal, thought I was gonna kick the bucket for sure.
mid 40's is where my heart usually sits when im sleeping. 60 when resting
I did come across this bit.
https://onlinelibrary.wiley.com/doi/10.1002/jmv.26515
We conducted a single-center hospital-based study and consecutively recruited 122 suspected and 104 confirmed patients with COVID-19 during January 24 to April 25, 2020. Clinical risk factors of COVID-19 were identified. The discriminative power of low calcium and phosphorus regarding the disease severity was evaluated. Low calcium and low phosphorus are more prevalent in severe or critical COVID-19 patients than moderate COVID-19 patients (odds ratio [OR], 15.07; 95% confidence interval [CI], 1.59–143.18 for calcium; OR, 6.90; 95% CI, 2.43–19.64 for phosphorus). The specificity in detecting the severe or critical patients among COVID-19 patients reached 98.5% (95% CI, 92.0%–99.7%) and 84.8% (95% CI, 74.3%–91.6%) by low calcium and low phosphorus, respectively, albeit with suboptimal sensitivity. Calcium and phosphorus combined with lymphocyte count could obtain the best discriminative performance for the severe COVID-19 patients (area under the curve [AUC] = 0.80), and combined with oxygenation index was promising (AUC = 0.71).
So, is the breathing problems causing low phosphorous, or is the low phosphorous causing the breathing problems?(worth mentioning that viral infections deplete phosphorous, as its crucial for most body functions)
However, it needs to be more elucidated by further studies whether hypophosphatemia in severe COVID-19 is directly related to COVID-19 or is just a complication of severe illness.
For me I could breathe during the day, but at 3 at night when your levels dip to their lowest, I thought I had fucking Chronic Obstruction Pulmonary Disorder from smoking weed, I didnt know what was going on. It wasnt until months later I demanded a kidney test and the phosphorous levels were actually .37mmol/l if I remember right this time.
In adults, the normal range is between 2.5 and 4.5 mg/dL (milligrams per deciliter). That equals roughly 0.8 – 1.45 mmol/L (millimoles per liter).
Once I discovered that and started drinking pop again and using phosphorous supplements I was able to slowly work it up to .80 after a year. Doctor tells me this is normal, so I stopped drinking pop and I cant take the supplements anymore because they are bound with sodium and taking one will give me cotton mouth for days, literally. You arent meant to take them forever, your supposed to fix the problem first, but these people here in canada are criminals and quacks.
Anyway. Come across this as well.
https://www.frontiersin.org/articles/10.3389/fimmu.2022.1006076/full
Results: We found that more than 85% of the COVID-19 infected patients presented a severe condition during the infection. In our sample, the mortality rate over 11-month follow was relatively low (8.4%) when compared to worldwide (approximately 36%). Long COVID was highly prevalent in COVID-19 survivors representing more than 80% of all cases. Phosphate and IL-10 were higher in the long COVID group, but only phosphate higher than 5.35 mg/dL appears to present an increased prevalence of long COVID, dyspnea, and fatigue.
You are proably thinking, if covid causes low phosphorous and is noted in most icu admissions, then why do people have high phosphorous from long covid?
https://journals.physiology.org/doi/full/10.1152/ajpendo.00474.2020
Symptoms observed in severe COVID-19 also fit well with those seen in classical hypophosphatemia and hypomagnesemia, such as thrombocytopenia, coagulopathy, dysfunction of liver and kidneys, neurologic disturbances, immunodeficiency, failure of heart and lungs, delayed weaning from a respirator, cardiac arrhythmia, seizures, and, finally, multiorgan failure. Deficiencies of phosphate and Mg can be amplified by kidney problems commonly observed in patients with COVID-19 resulting in their wastage into urine. Available data show that phosphate and Mg are deficient in COVID-19, with phosphate showing a remarkable correlation with its severity. In one experiment, patients with COVID-19 were supplemented with a cocktail of vitamin D3, Mg, and vitamin B12, with very encouraging results. We, thus, argue that patients with COVID-19 should be monitored and treated for phosphate and Mg deficiencies, ideally already in the early phases of infection. Supplementation of phosphate and Mg combined with vitamin D could also be implemented as a preventative strategy in populations at risk.
So, maybe im understanding this wrong, please feel free to share your thoughts as well.
Covid depletes phosphorous, so if your already low, you might die. People, like alcoholics, or people with something like crohn's, these people are walking around with not much phosphorous in them. For me i believe its the aki, the acute kidney injury I received during my lovely time at arca being poisoned with refrigerant. It seems this one specific part of my kidneys are busted, all other function is perfectly normal. If it was long covid, my calcium and magnesium should be out of whack, its perfectly normal. The only thing that is off for me is the phosphorous and the uric acid, which is handled by the same region in the kidney.
And once that happens, the phosphorous, the calcium, and the manganese dropping, this is what causes the organ damage is my best guess. Im no doctor.
But here is what gets me. They have known this since at least 2021, if not 2020. But whats the standard protocol?
Stick them on a ventilator, and give them something like Remdesivir, which will destroy your kidneys if you have any kind of kidney impairment because it cant metabolize it properly.
Remdesivir is not recommended for use in patients with an estimated glomerular filtration rate (eGFR) less than 30 mL/min1. 73 m2 owing to the presence of excipients3 that may accumulate in kidney dysfunction and worsen kidney or hepatic outcomes.
How many people were just straight up murdered by dr. fauci? Wasnt he the one who recommended this procedure?
I feel like some of this information, like this part.
Available data show that phosphate and Mg are deficient in COVID-19, with phosphate showing a remarkable correlation with its severity. In one experiment, patients with COVID-19 were supplemented with a cocktail of vitamin D3, Mg, and vitamin B12, with very encouraging results. We, thus, argue that patients with COVID-19 should be monitored and treated for phosphate and Mg deficiencies, ideally already in the early phases of infection. Supplementation of phosphate and Mg combined with vitamin D could also be implemented as a preventative strategy in populations at risk.
should be pinned in the sidebar somewhere.
I also wanted to mention something else, I was going to make my own thread but meh, ill say it again, I am a terrible op. I just dont care about much lol.
I was googling the relationship between long covid and low phosphorous. The last time I looked into this, was about 1 and 1/2 years ago when I had omicron, and passed out and my blood pressure was hitting something like 100/50, heart rate 32 while the emts had me hooked up. It was brutal, thought I was gonna kick the bucket for sure.
mid 40's is where my heart usually sits when im sleeping. 60 when resting
I did come across this bit.
https://onlinelibrary.wiley.com/doi/10.1002/jmv.26515
We conducted a single-center hospital-based study and consecutively recruited 122 suspected and 104 confirmed patients with COVID-19 during January 24 to April 25, 2020. Clinical risk factors of COVID-19 were identified. The discriminative power of low calcium and phosphorus regarding the disease severity was evaluated. Low calcium and low phosphorus are more prevalent in severe or critical COVID-19 patients than moderate COVID-19 patients (odds ratio [OR], 15.07; 95% confidence interval [CI], 1.59–143.18 for calcium; OR, 6.90; 95% CI, 2.43–19.64 for phosphorus). The specificity in detecting the severe or critical patients among COVID-19 patients reached 98.5% (95% CI, 92.0%–99.7%) and 84.8% (95% CI, 74.3%–91.6%) by low calcium and low phosphorus, respectively, albeit with suboptimal sensitivity. Calcium and phosphorus combined with lymphocyte count could obtain the best discriminative performance for the severe COVID-19 patients (area under the curve [AUC] = 0.80), and combined with oxygenation index was promising (AUC = 0.71).
So, is the breathing problems causing low phosphorous, or is the low phosphorous causing the breathing problems?(worth mentioning that viral infections deplete phosphorous, as its crucial for most body functions)
However, it needs to be more elucidated by further studies whether hypophosphatemia in severe COVID-19 is directly related to COVID-19 or is just a complication of severe illness.
For me I could breathe during the day, but at 3 at night when your levels dip to their lowest, I thought I had fucking Chronic Obstruction Pulmonary Disorder from smoking weed, I didnt know what was going on. It wasnt until months later I demanded a kidney test and the phosphorous levels were actually .37mmol/l if I remember right this time.
In adults, the normal range is between 2.5 and 4.5 mg/dL (milligrams per deciliter). That equals roughly 0.8 – 1.45 mmol/L (millimoles per liter).
Once I discovered that and started drinking pop again and using phosphorous supplements I was able to slowly work it up to .80 after a year. Doctor tells me this is normal, so I stopped drinking pop and I cant take the supplements anymore because they are bound with sodium and taking one will give me cotton mouth for days, literally. You arent meant to take them forever, your supposed to fix the problem first, but these people here in canada are criminals and quacks.
Anyway. Come across this as well.
https://www.frontiersin.org/articles/10.3389/fimmu.2022.1006076/full
Results: We found that more than 85% of the COVID-19 infected patients presented a severe condition during the infection. In our sample, the mortality rate over 11-month follow was relatively low (8.4%) when compared to worldwide (approximately 36%). Long COVID was highly prevalent in COVID-19 survivors representing more than 80% of all cases. Phosphate and IL-10 were higher in the long COVID group, but only phosphate higher than 5.35 mg/dL appears to present an increased prevalence of long COVID, dyspnea, and fatigue.
You are proably thinking, if covid causes low phosphorous and is noted in most icu admissions, then why do people have high phosphorous from long covid?
https://journals.physiology.org/doi/full/10.1152/ajpendo.00474.2020
Symptoms observed in severe COVID-19 also fit well with those seen in classical hypophosphatemia and hypomagnesemia, such as thrombocytopenia, coagulopathy, dysfunction of liver and kidneys, neurologic disturbances, immunodeficiency, failure of heart and lungs, delayed weaning from a respirator, cardiac arrhythmia, seizures, and, finally, multiorgan failure. Deficiencies of phosphate and Mg can be amplified by kidney problems commonly observed in patients with COVID-19 resulting in their wastage into urine. Available data show that phosphate and Mg are deficient in COVID-19, with phosphate showing a remarkable correlation with its severity. In one experiment, patients with COVID-19 were supplemented with a cocktail of vitamin D3, Mg, and vitamin B12, with very encouraging results. We, thus, argue that patients with COVID-19 should be monitored and treated for phosphate and Mg deficiencies, ideally already in the early phases of infection. Supplementation of phosphate and Mg combined with vitamin D could also be implemented as a preventative strategy in populations at risk.
So, maybe im understanding this wrong, please feel free to share your thoughts as well.
Covid depletes phosphorous, so if your already low, you might die. People, like alcoholics, or people with something like crohn's, these people are walking around with not much phosphorous in them. For me i believe its the aki, the acute kidney injury I received during my lovely time at arca being poisoned with refrigerant. It seems this one specific part of my kidneys are busted, all other function is perfectly normal. If it was long covid, my calcium and magnesium should be out of whack, its perfectly normal. The only thing that is off for me is the phosphorous and the uric acid, which is handled by the same region in the kidney. And once that happens, the calcium, and the manganese drop as well, this is what causes the organ damage is my best guess. Im no doctor.
But here is what gets me. They have known this since at least 2021, if not 2020. But whats the standard protocol?
Stick them on a ventilator, and give them something like Remdesivir, which will destroy your kidneys if you have any kind of kidney impairment because it cant metabolize it properly.
Remdesivir is not recommended for use in patients with an estimated glomerular filtration rate (eGFR) less than 30 mL/min1. 73 m2 owing to the presence of excipients3 that may accumulate in kidney dysfunction and worsen kidney or hepatic outcomes.
How many people were just straight up murdered by dr. fauci? Wasnt he the one who recommended this procedure?
I feel like some of this information, like this part.
Available data show that phosphate and Mg are deficient in COVID-19, with phosphate showing a remarkable correlation with its severity. In one experiment, patients with COVID-19 were supplemented with a cocktail of vitamin D3, Mg, and vitamin B12, with very encouraging results. We, thus, argue that patients with COVID-19 should be monitored and treated for phosphate and Mg deficiencies, ideally already in the early phases of infection. Supplementation of phosphate and Mg combined with vitamin D could also be implemented as a preventative strategy in populations at risk.
should be pinned in the sidebar somewhere.
I also wanted to mention something else, I was going to make my own thread but meh, ill say it again, I am a terrible op. I just dont care about much lol.
I was googling the relationship between long covid and low phosphorous. The last time I looked into this, was about 1 and 1/2 years ago when I had omicron, and passed out and my blood pressure was hitting something like 100/50, heart rate 32 while the emts had me hooked up. It was brutal, thought I was gonna kick the bucket for sure.
mid 40's is where my heart usually sits when im sleeping. 60 when resting
I did come across this bit.
https://onlinelibrary.wiley.com/doi/10.1002/jmv.26515
We conducted a single-center hospital-based study and consecutively recruited 122 suspected and 104 confirmed patients with COVID-19 during January 24 to April 25, 2020. Clinical risk factors of COVID-19 were identified. The discriminative power of low calcium and phosphorus regarding the disease severity was evaluated. Low calcium and low phosphorus are more prevalent in severe or critical COVID-19 patients than moderate COVID-19 patients (odds ratio [OR], 15.07; 95% confidence interval [CI], 1.59–143.18 for calcium; OR, 6.90; 95% CI, 2.43–19.64 for phosphorus). The specificity in detecting the severe or critical patients among COVID-19 patients reached 98.5% (95% CI, 92.0%–99.7%) and 84.8% (95% CI, 74.3%–91.6%) by low calcium and low phosphorus, respectively, albeit with suboptimal sensitivity. Calcium and phosphorus combined with lymphocyte count could obtain the best discriminative performance for the severe COVID-19 patients (area under the curve [AUC] = 0.80), and combined with oxygenation index was promising (AUC = 0.71).
So, is the breathing problems causing low phosphorous, or is the low phosphorous causing the breathing problems?(worth mentioning that viral infections deplete phosphorous, as its crucial for most body functions)
However, it needs to be more elucidated by further studies whether hypophosphatemia in severe COVID-19 is directly related to COVID-19 or is just a complication of severe illness.
For me I could breathe during the day, but at 3 at night when your levels dip to their lowest, I thought I had fucking Chronic Obstruction Pulmonary Disorder from smoking weed, I didnt know what was going on. It wasnt until months later I demanded a kidney test and the phosphorous levels were actually .37mmol/l if I remember right this time.
In adults, the normal range is between 2.5 and 4.5 mg/dL (milligrams per deciliter). That equals roughly 0.8 – 1.45 mmol/L (millimoles per liter).
Once I discovered that and started drinking pop again and using phosphorous supplements I was able to slowly work it up to .80 after a year. Doctor tells me this is normal, so I stopped drinking pop and I cant take the supplements anymore because they are bound with sodium and taking one will give me cotton mouth for days, literally. You arent meant to take them forever, your supposed to fix the problem first, but these people here in canada are criminals and quacks.
Anyway. Come across this as well.
https://www.frontiersin.org/articles/10.3389/fimmu.2022.1006076/full
Results: We found that more than 85% of the COVID-19 infected patients presented a severe condition during the infection. In our sample, the mortality rate over 11-month follow was relatively low (8.4%) when compared to worldwide (approximately 36%). Long COVID was highly prevalent in COVID-19 survivors representing more than 80% of all cases. Phosphate and IL-10 were higher in the long COVID group, but only phosphate higher than 5.35 mg/dL appears to present an increased prevalence of long COVID, dyspnea, and fatigue.
You are proably thinking, if covid causes low phosphorous and is noted in most icu admissions, then why do people have high phosphorous from long covid?
https://journals.physiology.org/doi/full/10.1152/ajpendo.00474.2020
Symptoms observed in severe COVID-19 also fit well with those seen in classical hypophosphatemia and hypomagnesemia, such as thrombocytopenia, coagulopathy, dysfunction of liver and kidneys, neurologic disturbances, immunodeficiency, failure of heart and lungs, delayed weaning from a respirator, cardiac arrhythmia, seizures, and, finally, multiorgan failure. Deficiencies of phosphate and Mg can be amplified by kidney problems commonly observed in patients with COVID-19 resulting in their wastage into urine. Available data show that phosphate and Mg are deficient in COVID-19, with phosphate showing a remarkable correlation with its severity. In one experiment, patients with COVID-19 were supplemented with a cocktail of vitamin D3, Mg, and vitamin B12, with very encouraging results. We, thus, argue that patients with COVID-19 should be monitored and treated for phosphate and Mg deficiencies, ideally already in the early phases of infection. Supplementation of phosphate and Mg combined with vitamin D could also be implemented as a preventative strategy in populations at risk.
So, maybe im understanding this wrong, please feel free to share your thoughts as well.
Covid depletes phosphorous, so if your already low, you might die. People, like alcoholics, or people with something like crohn's, these people are walking around with not much phosphorous in them. For me i believe its the aki, the acute kidney injury I received during my lovely time at arca being poisoned with refrigerant. It seems this one specific part of my kidneys are busted, all other function is perfectly normal. If it was long covid, my calcium and magnesium should be out of whack, its perfectly normal. The only thing that is off for me is the phosphorous and the uric acid, which is handled by the same region in the kidney.
But here is what gets me. They have known this since at least 2021, if not 2020. But whats the standard protocol?
Stick them on a ventilator, and give them something like Remdesivir, which will destroy your kidneys if you have any kind of kidney impairment because it cant metabolize it properly.
Remdesivir is not recommended for use in patients with an estimated glomerular filtration rate (eGFR) less than 30 mL/min1. 73 m2 owing to the presence of excipients3 that may accumulate in kidney dysfunction and worsen kidney or hepatic outcomes.
How many people were just straight up murdered by dr. fauci? Wasnt he the one who recommended this procedure?
I feel like some of this information, like this part.
Available data show that phosphate and Mg are deficient in COVID-19, with phosphate showing a remarkable correlation with its severity. In one experiment, patients with COVID-19 were supplemented with a cocktail of vitamin D3, Mg, and vitamin B12, with very encouraging results. We, thus, argue that patients with COVID-19 should be monitored and treated for phosphate and Mg deficiencies, ideally already in the early phases of infection. Supplementation of phosphate and Mg combined with vitamin D could also be implemented as a preventative strategy in populations at risk.
should be pinned in the sidebar somewhere.
I also wanted to mention something, I was going to make my own thread but meh, ill say it again, I am a terrible op. I just dont care about much lol.
I was googling the relationship between long covid and low phosphorous. The last time I looked into this, was about 1 and 1/2 years ago when I had omicron, and passed out and my blood pressure was hitting something like 100/50, heart rate 32 while the emts had me hooked up. It was brutal, thought I was gonna kick the bucket for sure.
mid 40's is where my heart usually sits when im sleeping. 60 when resting
I did come across this bit.
https://onlinelibrary.wiley.com/doi/10.1002/jmv.26515
We conducted a single-center hospital-based study and consecutively recruited 122 suspected and 104 confirmed patients with COVID-19 during January 24 to April 25, 2020. Clinical risk factors of COVID-19 were identified. The discriminative power of low calcium and phosphorus regarding the disease severity was evaluated. Low calcium and low phosphorus are more prevalent in severe or critical COVID-19 patients than moderate COVID-19 patients (odds ratio [OR], 15.07; 95% confidence interval [CI], 1.59–143.18 for calcium; OR, 6.90; 95% CI, 2.43–19.64 for phosphorus). The specificity in detecting the severe or critical patients among COVID-19 patients reached 98.5% (95% CI, 92.0%–99.7%) and 84.8% (95% CI, 74.3%–91.6%) by low calcium and low phosphorus, respectively, albeit with suboptimal sensitivity. Calcium and phosphorus combined with lymphocyte count could obtain the best discriminative performance for the severe COVID-19 patients (area under the curve [AUC] = 0.80), and combined with oxygenation index was promising (AUC = 0.71).
So, is the breathing problems causing low phosphorous, or is the low phosphorous causing the breathing problems?(worth mentioning that viral infections deplete phosphorous, as its crucial for most body functions)
However, it needs to be more elucidated by further studies whether hypophosphatemia in severe COVID-19 is directly related to COVID-19 or is just a complication of severe illness.
For me I could breathe during the day, but at 3 at night when your levels dip to their lowest, I thought I had fucking Chronic Obstruction Pulmonary Disorder from smoking weed, I didnt know what was going on. It wasnt until months later I demanded a kidney test and the phosphorous levels were actually .37mmol/l if I remember right this time.
In adults, the normal range is between 2.5 and 4.5 mg/dL (milligrams per deciliter). That equals roughly 0.8 – 1.45 mmol/L (millimoles per liter).
Once I discovered that and started drinking pop again and using phosphorous supplements I was able to slowly work it up to .80 after a year. Doctor tells me this is normal, so I stopped drinking pop and I cant take the supplements anymore because they are bound with sodium and taking one will give me cotton mouth for days, literally. You arent meant to take them forever, your supposed to fix the problem first, but these people here in canada are criminals and quacks.
Anyway. Come across this as well.
https://www.frontiersin.org/articles/10.3389/fimmu.2022.1006076/full
Results: We found that more than 85% of the COVID-19 infected patients presented a severe condition during the infection. In our sample, the mortality rate over 11-month follow was relatively low (8.4%) when compared to worldwide (approximately 36%). Long COVID was highly prevalent in COVID-19 survivors representing more than 80% of all cases. Phosphate and IL-10 were higher in the long COVID group, but only phosphate higher than 5.35 mg/dL appears to present an increased prevalence of long COVID, dyspnea, and fatigue.
You are proably thinking, if covid causes low phosphorous and is noted in most icu admissions, then why do people have high phosphorous from long covid?
https://journals.physiology.org/doi/full/10.1152/ajpendo.00474.2020
Symptoms observed in severe COVID-19 also fit well with those seen in classical hypophosphatemia and hypomagnesemia, such as thrombocytopenia, coagulopathy, dysfunction of liver and kidneys, neurologic disturbances, immunodeficiency, failure of heart and lungs, delayed weaning from a respirator, cardiac arrhythmia, seizures, and, finally, multiorgan failure. Deficiencies of phosphate and Mg can be amplified by kidney problems commonly observed in patients with COVID-19 resulting in their wastage into urine. Available data show that phosphate and Mg are deficient in COVID-19, with phosphate showing a remarkable correlation with its severity. In one experiment, patients with COVID-19 were supplemented with a cocktail of vitamin D3, Mg, and vitamin B12, with very encouraging results. We, thus, argue that patients with COVID-19 should be monitored and treated for phosphate and Mg deficiencies, ideally already in the early phases of infection. Supplementation of phosphate and Mg combined with vitamin D could also be implemented as a preventative strategy in populations at risk.
So, maybe im understanding this wrong, please feel free to share your thoughts as well.
Covid depletes phosphorous, so if your already low, you might die. People, like alcoholics, or people with something like crohn's, these people are walking around with not much phosphorous in them. For me i believe its the aki, the acute kidney injury I received during my lovely time at arca being poisoned with refrigerant. It seems this one specific part of my kidneys are busted, all other function is perfectly normal. If it was long covid, my calcium and magnesium should be out of whack, its perfectly normal. The only thing that is off for me is the phosphorous and the uric acid, which is handled by the same region in the kidney.
But here is what gets me. They have known this since at least 2021, if not 2020. But whats the standard protocol?
Stick them on a ventilator, and give them something like Remdesivir, which will destroy your kidneys if you have any kind of kidney impairment because it cant metabolize it properly.
Remdesivir is not recommended for use in patients with an estimated glomerular filtration rate (eGFR) less than 30 mL/min1. 73 m2 owing to the presence of excipients3 that may accumulate in kidney dysfunction and worsen kidney or hepatic outcomes.
How many people were just straight up murdered by dr. fauci? Wasnt he the one who recommended this procedure?
I feel like some of this information, like this part.
Available data show that phosphate and Mg are deficient in COVID-19, with phosphate showing a remarkable correlation with its severity. In one experiment, patients with COVID-19 were supplemented with a cocktail of vitamin D3, Mg, and vitamin B12, with very encouraging results. We, thus, argue that patients with COVID-19 should be monitored and treated for phosphate and Mg deficiencies, ideally already in the early phases of infection. Supplementation of phosphate and Mg combined with vitamin D could also be implemented as a preventative strategy in populations at risk.
should be pinned in the sidebar somewhere.
I also wanted to mention something, I was going to make my own thread but meh, ill say it again, I am a terrible op. I just dont care about much lol.
I was googling the relationship between long covid and low phosphorous. The last time I looked into this, was about 1 and 1/2 years ago when I had omicron, and passed out and my blood pressure was hitting something like 100/50, heart rate 32 while the emts had me hooked up. It was brutal, thought I was gonna kick the bucket for sure.
mid 40's is where my heart usually sits when im sleeping. 60 when resting
I did come across this bit.
https://onlinelibrary.wiley.com/doi/10.1002/jmv.26515
We conducted a single-center hospital-based study and consecutively recruited 122 suspected and 104 confirmed patients with COVID-19 during January 24 to April 25, 2020. Clinical risk factors of COVID-19 were identified. The discriminative power of low calcium and phosphorus regarding the disease severity was evaluated. Low calcium and low phosphorus are more prevalent in severe or critical COVID-19 patients than moderate COVID-19 patients (odds ratio [OR], 15.07; 95% confidence interval [CI], 1.59–143.18 for calcium; OR, 6.90; 95% CI, 2.43–19.64 for phosphorus). The specificity in detecting the severe or critical patients among COVID-19 patients reached 98.5% (95% CI, 92.0%–99.7%) and 84.8% (95% CI, 74.3%–91.6%) by low calcium and low phosphorus, respectively, albeit with suboptimal sensitivity. Calcium and phosphorus combined with lymphocyte count could obtain the best discriminative performance for the severe COVID-19 patients (area under the curve [AUC] = 0.80), and combined with oxygenation index was promising (AUC = 0.71).
So, is the breathing problems causing low phosphorous, or is the low phosphorous causing the breathing problems? For me I could breathe during the day, but at 3 at night when your levels dip to their lowest, I thought I had fucking Chronic Obstruction Pulmonary Disorder from smoking weed, I didnt know what was going on. It wasnt until months later I demanded a kidney test and the phosphorous levels were actually .37mmol/l if I remember right this time.
In adults, the normal range is between 2.5 and 4.5 mg/dL (milligrams per deciliter). That equals roughly 0.8 – 1.45 mmol/L (millimoles per liter).
Once I discovered that and started drinking pop again and using phosphorous supplements I was able to slowly work it up to .80 after a year. Doctor tells me this is normal, so I stopped drinking pop and I cant take the supplements anymore because they are bound with sodium and taking one will give me cotton mouth for days, literally. You arent meant to take them forever, your supposed to fix the problem first, but these people here in canada are criminals and quacks.
Anyway. Come across this as well.
https://www.frontiersin.org/articles/10.3389/fimmu.2022.1006076/full
Results: We found that more than 85% of the COVID-19 infected patients presented a severe condition during the infection. In our sample, the mortality rate over 11-month follow was relatively low (8.4%) when compared to worldwide (approximately 36%). Long COVID was highly prevalent in COVID-19 survivors representing more than 80% of all cases. Phosphate and IL-10 were higher in the long COVID group, but only phosphate higher than 5.35 mg/dL appears to present an increased prevalence of long COVID, dyspnea, and fatigue.
You are proably thinking, if covid causes low phosphorous and is noted in most icu admissions, then why do people have high phosphorous from long covid?
https://journals.physiology.org/doi/full/10.1152/ajpendo.00474.2020
Symptoms observed in severe COVID-19 also fit well with those seen in classical hypophosphatemia and hypomagnesemia, such as thrombocytopenia, coagulopathy, dysfunction of liver and kidneys, neurologic disturbances, immunodeficiency, failure of heart and lungs, delayed weaning from a respirator, cardiac arrhythmia, seizures, and, finally, multiorgan failure. Deficiencies of phosphate and Mg can be amplified by kidney problems commonly observed in patients with COVID-19 resulting in their wastage into urine. Available data show that phosphate and Mg are deficient in COVID-19, with phosphate showing a remarkable correlation with its severity. In one experiment, patients with COVID-19 were supplemented with a cocktail of vitamin D3, Mg, and vitamin B12, with very encouraging results. We, thus, argue that patients with COVID-19 should be monitored and treated for phosphate and Mg deficiencies, ideally already in the early phases of infection. Supplementation of phosphate and Mg combined with vitamin D could also be implemented as a preventative strategy in populations at risk.
So, maybe im understanding this wrong, please feel free to share your thoughts as well.
Covid depletes phosphorous, so if your already low, you might die. People, like alcoholics, or people with something like crohn's, these people are walking around with not much phosphorous in them. For me i believe its the aki, the acute kidney injury I received during my lovely time at arca being poisoned with refrigerant. It seems this one specific part of my kidneys are busted, all other function is perfectly normal. If it was long covid, my calcium and magnesium should be out of whack, its perfectly normal. The only thing that is off for me is the phosphorous and the uric acid, which is handled by the same region in the kidney.
But here is what gets me. They have known this since at least 2021, if not 2020. But whats the standard protocol?
Stick them on a ventilator, and give them something like Remdesivir, which will destroy your kidneys if you have any kind of kidney impairment because it cant metabolize it properly.
Remdesivir is not recommended for use in patients with an estimated glomerular filtration rate (eGFR) less than 30 mL/min1. 73 m2 owing to the presence of excipients3 that may accumulate in kidney dysfunction and worsen kidney or hepatic outcomes.
How many people were just straight up murdered by dr. fauci? Wasnt he the one who recommended this procedure?
I feel like some of this information, like this part.
Available data show that phosphate and Mg are deficient in COVID-19, with phosphate showing a remarkable correlation with its severity. In one experiment, patients with COVID-19 were supplemented with a cocktail of vitamin D3, Mg, and vitamin B12, with very encouraging results. We, thus, argue that patients with COVID-19 should be monitored and treated for phosphate and Mg deficiencies, ideally already in the early phases of infection. Supplementation of phosphate and Mg combined with vitamin D could also be implemented as a preventative strategy in populations at risk.
should be pinned in the sidebar somewhere.
I also wanted to mention something, I was going to make my own thread but meh, ill say it again, I am a terrible op. I just dont care about much lol.
I was googling the relationship between long covid and low phosphorous. The last time I looked into this, was about 1 and 1/2 years ago when I had omicron, and passed out and my blood pressure was hitting something like 100/50, heart rate 32 while the emts had me hooked up. It was brutal, thought I was gonna kick the bucket for sure.
mid 40's is where my heart usually sits when im sleeping. 60 when resting
I did come across this bit.
https://onlinelibrary.wiley.com/doi/10.1002/jmv.26515
We conducted a single-center hospital-based study and consecutively recruited 122 suspected and 104 confirmed patients with COVID-19 during January 24 to April 25, 2020. Clinical risk factors of COVID-19 were identified. The discriminative power of low calcium and phosphorus regarding the disease severity was evaluated. Low calcium and low phosphorus are more prevalent in severe or critical COVID-19 patients than moderate COVID-19 patients (odds ratio [OR], 15.07; 95% confidence interval [CI], 1.59–143.18 for calcium; OR, 6.90; 95% CI, 2.43–19.64 for phosphorus). The specificity in detecting the severe or critical patients among COVID-19 patients reached 98.5% (95% CI, 92.0%–99.7%) and 84.8% (95% CI, 74.3%–91.6%) by low calcium and low phosphorus, respectively, albeit with suboptimal sensitivity. Calcium and phosphorus combined with lymphocyte count could obtain the best discriminative performance for the severe COVID-19 patients (area under the curve [AUC] = 0.80), and combined with oxygenation index was promising (AUC = 0.71).
So, is the breathing problems causing low phosphorous, or is the low phosphorous causing the breathing problems? For me I could breathe during the day, but at 3 at night when your levels dip to their lowest, I thought I had fucking Chronic Obstruction Pulmonary Disorder from smoking weed, I didnt know what was going on. It wasnt until months later I demanded a kidney test and the phosphorous levels were actually .37mmol/l if I remember right this time.
In adults, the normal range is between 2.5 and 4.5 mg/dL (milligrams per deciliter). That equals roughly 0.8 – 1.45 mmol/L (millimoles per liter).
Once I discovered that and started drinking pop again and using phosphorous supplements I was able to slowly work it up to .80 after a year. Doctor tells me this is normal, so I stopped drinking pop and I cant take the supplements anymore because they are bound with sodium and taking one will give me cotton mouth for days, literally. You arent meant to take them forever, your supposed to fix the problem first, but these people here in canada are criminals and quacks.
Anyway. Come across this as well.
https://www.frontiersin.org/articles/10.3389/fimmu.2022.1006076/full
Results: We found that more than 85% of the COVID-19 infected patients presented a severe condition during the infection. In our sample, the mortality rate over 11-month follow was relatively low (8.4%) when compared to worldwide (approximately 36%). Long COVID was highly prevalent in COVID-19 survivors representing more than 80% of all cases. Phosphate and IL-10 were higher in the long COVID group, but only phosphate higher than 5.35 mg/dL appears to present an increased prevalence of long COVID, dyspnea, and fatigue.
How does it cause hyperphosphatemia if low phosphorous is noted in most icu admissions?
https://journals.physiology.org/doi/full/10.1152/ajpendo.00474.2020
Symptoms observed in severe COVID-19 also fit well with those seen in classical hypophosphatemia and hypomagnesemia, such as thrombocytopenia, coagulopathy, dysfunction of liver and kidneys, neurologic disturbances, immunodeficiency, failure of heart and lungs, delayed weaning from a respirator, cardiac arrhythmia, seizures, and, finally, multiorgan failure. Deficiencies of phosphate and Mg can be amplified by kidney problems commonly observed in patients with COVID-19 resulting in their wastage into urine. Available data show that phosphate and Mg are deficient in COVID-19, with phosphate showing a remarkable correlation with its severity. In one experiment, patients with COVID-19 were supplemented with a cocktail of vitamin D3, Mg, and vitamin B12, with very encouraging results. We, thus, argue that patients with COVID-19 should be monitored and treated for phosphate and Mg deficiencies, ideally already in the early phases of infection. Supplementation of phosphate and Mg combined with vitamin D could also be implemented as a preventative strategy in populations at risk.
So, maybe im understanding this wrong, please feel free to share your thoughts as well.
Covid depletes phosphorous, so if your already low, you might die. People, like alcoholics, or people with something like crohn's, these people are walking around with not much phosphorous in them. For me i believe its the aki, the acute kidney injury I received during my lovely time at arca being poisoned with refrigerant. It seems this one specific part of my kidneys are busted, all other function is perfectly normal. If it was long covid, my calcium and magnesium should be out of whack, its perfectly normal. The only thing that is off for me is the phosphorous and the uric acid, which is handled by the same region in the kidney.
But here is what gets me. They have known this since at least 2021, if not 2020. But whats the standard protocol?
Stick them on a ventilator, and give them something like Remdesivir, which will destroy your kidneys if you have any kind of kidney impairment because it cant metabolize it properly.
Remdesivir is not recommended for use in patients with an estimated glomerular filtration rate (eGFR) less than 30 mL/min1. 73 m2 owing to the presence of excipients3 that may accumulate in kidney dysfunction and worsen kidney or hepatic outcomes.
How many people were just straight up murdered by dr. fauci? Wasnt he the one who recommended this procedure?
I feel like some of this information, like this part.
Available data show that phosphate and Mg are deficient in COVID-19, with phosphate showing a remarkable correlation with its severity. In one experiment, patients with COVID-19 were supplemented with a cocktail of vitamin D3, Mg, and vitamin B12, with very encouraging results. We, thus, argue that patients with COVID-19 should be monitored and treated for phosphate and Mg deficiencies, ideally already in the early phases of infection. Supplementation of phosphate and Mg combined with vitamin D could also be implemented as a preventative strategy in populations at risk.
should be pinned in the sidebar somewhere.
I also wanted to mention something, I was going to make my own thread but meh, ill say it again, I am a terrible op. I just dont care about much lol.
I was googling the relationship between long covid and low phosphorous. The last time I looked into this, was about 1 and 1/2 years ago when I had omicron, and passed out and my blood pressure was hitting something like 100/50, heart rate 32 while the emts had me hooked up. It was brutal, thought I was gonna kick the bucket for sure.
mid 40's is where my heart usually sits when im sleeping. 60 when resting
I did come across this bit.
https://onlinelibrary.wiley.com/doi/10.1002/jmv.26515
We conducted a single-center hospital-based study and consecutively recruited 122 suspected and 104 confirmed patients with COVID-19 during January 24 to April 25, 2020. Clinical risk factors of COVID-19 were identified. The discriminative power of low calcium and phosphorus regarding the disease severity was evaluated. Low calcium and low phosphorus are more prevalent in severe or critical COVID-19 patients than moderate COVID-19 patients (odds ratio [OR], 15.07; 95% confidence interval [CI], 1.59–143.18 for calcium; OR, 6.90; 95% CI, 2.43–19.64 for phosphorus). The specificity in detecting the severe or critical patients among COVID-19 patients reached 98.5% (95% CI, 92.0%–99.7%) and 84.8% (95% CI, 74.3%–91.6%) by low calcium and low phosphorus, respectively, albeit with suboptimal sensitivity. Calcium and phosphorus combined with lymphocyte count could obtain the best discriminative performance for the severe COVID-19 patients (area under the curve [AUC] = 0.80), and combined with oxygenation index was promising (AUC = 0.71).
So, is the breathing problems causing low phosphorous, or is the low phosphorous causing the breathing problems? For me I could breathe during the day, but at 3 at night when your levels dip to their lowest, I thought I had fucking Chronic Obstruction Pulmonary Disorder from smoking weed, I didnt know what was going on. It wasnt until months later I demanded a kidney test and the phosphorous levels were actually .37mmol/l if I remember right this time.
In adults, the normal range is between 2.5 and 4.5 mg/dL (milligrams per deciliter). That equals roughly 0.8 – 1.45 mmol/L (millimoles per liter).
Once I discovered that and started drinking pop again and using phosphorous supplements I was able to slowly work it up to .80 after a year. Doctor tells me this is normal, so I stopped drinking pop and I cant take the supplements anymore because they are bound with sodium and taking one will give me cotton mouth for days, literally. You arent meant to take them forever, your supposed to fix the problem first, but these people here in canada are criminals and quacks.
Anyway. Come across this as well.
https://www.frontiersin.org/articles/10.3389/fimmu.2022.1006076/full
Results: We found that more than 85% of the COVID-19 infected patients presented a severe condition during the infection. In our sample, the mortality rate over 11-month follow was relatively low (8.4%) when compared to worldwide (approximately 36%). Long COVID was highly prevalent in COVID-19 survivors representing more than 80% of all cases. Phosphate and IL-10 were higher in the long COVID group, but only phosphate higher than 5.35 mg/dL appears to present an increased prevalence of long COVID, dyspnea, and fatigue.
How does it cause hyperphosphatemia if low phosphorous is noted in most icu admissions?
https://journals.physiology.org/doi/full/10.1152/ajpendo.00474.2020
Symptoms observed in severe COVID-19 also fit well with those seen in classical hypophosphatemia and hypomagnesemia, such as thrombocytopenia, coagulopathy, dysfunction of liver and kidneys, neurologic disturbances, immunodeficiency, failure of heart and lungs, delayed weaning from a respirator, cardiac arrhythmia, seizures, and, finally, multiorgan failure. Deficiencies of phosphate and Mg can be amplified by kidney problems commonly observed in patients with COVID-19 resulting in their wastage into urine. Available data show that phosphate and Mg are deficient in COVID-19, with phosphate showing a remarkable correlation with its severity. In one experiment, patients with COVID-19 were supplemented with a cocktail of vitamin D3, Mg, and vitamin B12, with very encouraging results. We, thus, argue that patients with COVID-19 should be monitored and treated for phosphate and Mg deficiencies, ideally already in the early phases of infection. Supplementation of phosphate and Mg combined with vitamin D could also be implemented as a preventative strategy in populations at risk.
So, maybe im understanding this wrong, please feel free to share your thoughts as well.
Covid depletes phosphorous, so if your already low, you might die. People, like alcoholics, or people with something like crohn's, these people are walking around with not much phosphorous in them. For me i believe its the aki, the acute kidney injury I received during my lovely time at arca being poisoned with refrigerant. It seems this one specific part of my kidneys are busted, all other function is perfectly normal. If it was long covid, my calcium and magnesium should be out of whack, its perfectly normal. The only thing that is off for me is the phosphorous and the uric acid, which is handled by the same region in the kidney.
But here is what gets me. They have known this since at least 2021, if not 2020. But whats the standard protocol?
Stick them on a ventilator, and give them something Remdesivir , which will destroy your kidneys if you have any kind of kidney impairment because it cant metabolize it properly.
Remdesivir is not recommended for use in patients with an estimated glomerular filtration rate (eGFR) less than 30 mL/min1. 73 m2 owing to the presence of excipients3 that may accumulate in kidney dysfunction and worsen kidney or hepatic outcomes.
How many people were just straight up murdered by dr. fauci? Wasnt he the one who recommended this procedure?
I feel like some of this information, like this part.
Available data show that phosphate and Mg are deficient in COVID-19, with phosphate showing a remarkable correlation with its severity. In one experiment, patients with COVID-19 were supplemented with a cocktail of vitamin D3, Mg, and vitamin B12, with very encouraging results. We, thus, argue that patients with COVID-19 should be monitored and treated for phosphate and Mg deficiencies, ideally already in the early phases of infection. Supplementation of phosphate and Mg combined with vitamin D could also be implemented as a preventative strategy in populations at risk.
should be pinned in the sidebar somewhere.
I also wanted to mention something, I was going to make my own thread but meh, ill say it again, I am a terrible op. I just dont care about much lol.
I was googling the relationship between long covid and low phosphorous. The last time I looked into this, was about 1 and 1/2 years ago when I had omicron, and passed out and my blood pressure was hitting something like 100/50, heart rate 32 while the emts had me hooked up. It was brutal, thought I was gonna kick the bucket for sure.
mid 40's is where my heart usually sits when im sleeping. 60 when resting
I did come across this bit.
https://onlinelibrary.wiley.com/doi/10.1002/jmv.26515
We conducted a single-center hospital-based study and consecutively recruited 122 suspected and 104 confirmed patients with COVID-19 during January 24 to April 25, 2020. Clinical risk factors of COVID-19 were identified. The discriminative power of low calcium and phosphorus regarding the disease severity was evaluated. Low calcium and low phosphorus are more prevalent in severe or critical COVID-19 patients than moderate COVID-19 patients (odds ratio [OR], 15.07; 95% confidence interval [CI], 1.59–143.18 for calcium; OR, 6.90; 95% CI, 2.43–19.64 for phosphorus). The specificity in detecting the severe or critical patients among COVID-19 patients reached 98.5% (95% CI, 92.0%–99.7%) and 84.8% (95% CI, 74.3%–91.6%) by low calcium and low phosphorus, respectively, albeit with suboptimal sensitivity. Calcium and phosphorus combined with lymphocyte count could obtain the best discriminative performance for the severe COVID-19 patients (area under the curve [AUC] = 0.80), and combined with oxygenation index was promising (AUC = 0.71).
So, is the breathing problems causing low phosphorous, or is the low phosphorous causing the breathing problems? For me I could breathe during the day, but at 3 at night when your levels dip to their lowest, I thought I had fucking Chronic Obstruction Pulmonary Disorder from smoking weed, I didnt know what was going on. It wasnt until months later I demanded a kidney test and the phosphorous levels were actually .37mmol/l if I remember right this time.
In adults, the normal range is between 2.5 and 4.5 mg/dL (milligrams per deciliter). That equals roughly 0.8 – 1.45 mmol/L (millimoles per liter).
Once I discovered that and started drinking pop again and using phosphorous supplements I was able to slowly work it up to .80 after a year. Doctor tells me this is normal, so I stopped drinking pop and I cant take the supplements anymore because they are bound with sodium and taking one will give me cotton mouth for days, literally. You arent meant to take them forever, your supposed to fix the problem first, but these people here in canada are criminals and quacks.
Anyway. Come across this as well.
https://www.frontiersin.org/articles/10.3389/fimmu.2022.1006076/full
Results: We found that more than 85% of the COVID-19 infected patients presented a severe condition during the infection. In our sample, the mortality rate over 11-month follow was relatively low (8.4%) when compared to worldwide (approximately 36%). Long COVID was highly prevalent in COVID-19 survivors representing more than 80% of all cases. Phosphate and IL-10 were higher in the long COVID group, but only phosphate higher than 5.35 mg/dL appears to present an increased prevalence of long COVID, dyspnea, and fatigue.
How does it cause hyperphosphatemia if low phosphorous is noted in most icu admissions?
https://journals.physiology.org/doi/full/10.1152/ajpendo.00474.2020
Symptoms observed in severe COVID-19 also fit well with those seen in classical hypophosphatemia and hypomagnesemia, such as thrombocytopenia, coagulopathy, dysfunction of liver and kidneys, neurologic disturbances, immunodeficiency, failure of heart and lungs, delayed weaning from a respirator, cardiac arrhythmia, seizures, and, finally, multiorgan failure. Deficiencies of phosphate and Mg can be amplified by kidney problems commonly observed in patients with COVID-19 resulting in their wastage into urine. Available data show that phosphate and Mg are deficient in COVID-19, with phosphate showing a remarkable correlation with its severity. In one experiment, patients with COVID-19 were supplemented with a cocktail of vitamin D3, Mg, and vitamin B12, with very encouraging results. We, thus, argue that patients with COVID-19 should be monitored and treated for phosphate and Mg deficiencies, ideally already in the early phases of infection. Supplementation of phosphate and Mg combined with vitamin D could also be implemented as a preventative strategy in populations at risk.
So, maybe im understanding this wrong, please feel free to share your thoughts as well.
Covid depletes phosphorous, so if your already low, you might die. People, like alcoholics, or people with something like crohn's, these people are walking around with not much phosphorous in them. For me i believe its the aki, the acute kidney injury I received during my lovely time at arca being poisoned with refrigerant. It seems this one specific part of my kidneys are busted, all other function is perfectly normal. If it was long covid, everything would be out of whack, like calcium, magnesium, vitamin d, the only thing that is off for me is the phosphorous and the uric acid, which is handled by the same region in the kidney.
But here is what gets me. They have known this since at least 2021, if not 2020. But whats the standard protocol?
Stick them on a ventilator, and give them something Remdesivir , which will destroy your kidneys if you have any kind of kidney impairment because it cant metabolize it properly.
Remdesivir is not recommended for use in patients with an estimated glomerular filtration rate (eGFR) less than 30 mL/min1. 73 m2 owing to the presence of excipients3 that may accumulate in kidney dysfunction and worsen kidney or hepatic outcomes.
How many people were just straight up murdered by dr. fauci? Wasnt he the one who recommended this procedure?
I feel like some of this information, like this part.
Available data show that phosphate and Mg are deficient in COVID-19, with phosphate showing a remarkable correlation with its severity. In one experiment, patients with COVID-19 were supplemented with a cocktail of vitamin D3, Mg, and vitamin B12, with very encouraging results. We, thus, argue that patients with COVID-19 should be monitored and treated for phosphate and Mg deficiencies, ideally already in the early phases of infection. Supplementation of phosphate and Mg combined with vitamin D could also be implemented as a preventative strategy in populations at risk.
should be pinned in the sidebar somewhere.
I also wanted to mention something, I was going to make my own thread but meh, ill say it again, I am a terrible op. I just dont care about much lol.
I was googling the relationship between long covid and low phosphorous. The last time I looked into this, was about 1 and 1/2 years ago when I had omicron, and passed out and my blood pressure was hitting something like 100/50, heart rate 32 while the emts had me hooked up. It was brutal, thought I was gonna kick the bucket for sure.
mid 40's is where my heart usually sits when im sleeping. 60 when resting
I did come across this bit.
https://onlinelibrary.wiley.com/doi/10.1002/jmv.26515
We conducted a single-center hospital-based study and consecutively recruited 122 suspected and 104 confirmed patients with COVID-19 during January 24 to April 25, 2020. Clinical risk factors of COVID-19 were identified. The discriminative power of low calcium and phosphorus regarding the disease severity was evaluated. Low calcium and low phosphorus are more prevalent in severe or critical COVID-19 patients than moderate COVID-19 patients (odds ratio [OR], 15.07; 95% confidence interval [CI], 1.59–143.18 for calcium; OR, 6.90; 95% CI, 2.43–19.64 for phosphorus). The specificity in detecting the severe or critical patients among COVID-19 patients reached 98.5% (95% CI, 92.0%–99.7%) and 84.8% (95% CI, 74.3%–91.6%) by low calcium and low phosphorus, respectively, albeit with suboptimal sensitivity. Calcium and phosphorus combined with lymphocyte count could obtain the best discriminative performance for the severe COVID-19 patients (area under the curve [AUC] = 0.80), and combined with oxygenation index was promising (AUC = 0.71).
So, is the breathing problems causing low phosphorous, or is the low phosphorous causing the breathing problems? For me I could breathe during the day, but at 3 at night when your levels dip to their lowest, I thought I had fucking Chronic Obstruction Pulmonary Disorder from smoking weed, I didnt know what was going on. It wasnt until months later I demanded a kidney test and the phosphorous levels were actually .37mmol/l if I remember right this time.
In adults, the normal range is between 2.5 and 4.5 mg/dL (milligrams per deciliter). That equals roughly 0.8 – 1.45 mmol/L (millimoles per liter).
Once I discovered that and started drinking pop again and using phosphorous supplements I was able to slowly work it up to .80 after a year. Doctor tells me this is normal, so I stopped drinking pop and I cant take the supplements anymore because they are bound with sodium and taking one will give me cotton mouth for days, literally. You arent meant to take them forever, your supposed to fix the problem first, but these people here in canada are criminals and quacks.
Anyway. Come across this as well.
https://www.frontiersin.org/articles/10.3389/fimmu.2022.1006076/full
Results: We found that more than 85% of the COVID-19 infected patients presented a severe condition during the infection. In our sample, the mortality rate over 11-month follow was relatively low (8.4%) when compared to worldwide (approximately 36%). Long COVID was highly prevalent in COVID-19 survivors representing more than 80% of all cases. Phosphate and IL-10 were higher in the long COVID group, but only phosphate higher than 5.35 mg/dL appears to present an increased prevalence of long COVID, dyspnea, and fatigue.
How does it cause hyperphosphatemia if low phosphorous is noted in most icu admissions?
https://journals.physiology.org/doi/full/10.1152/ajpendo.00474.2020
Symptoms observed in severe COVID-19 also fit well with those seen in classical hypophosphatemia and hypomagnesemia, such as thrombocytopenia, coagulopathy, dysfunction of liver and kidneys, neurologic disturbances, immunodeficiency, failure of heart and lungs, delayed weaning from a respirator, cardiac arrhythmia, seizures, and, finally, multiorgan failure. Deficiencies of phosphate and Mg can be amplified by kidney problems commonly observed in patients with COVID-19 resulting in their wastage into urine. Available data show that phosphate and Mg are deficient in COVID-19, with phosphate showing a remarkable correlation with its severity. In one experiment, patients with COVID-19 were supplemented with a cocktail of vitamin D3, Mg, and vitamin B12, with very encouraging results. We, thus, argue that patients with COVID-19 should be monitored and treated for phosphate and Mg deficiencies, ideally already in the early phases of infection. Supplementation of phosphate and Mg combined with vitamin D could also be implemented as a preventative strategy in populations at risk.
So, maybe im understanding this wrong, please feel free to share your thoughts as well.
Covid depletes phosphorous, so if your already low, you might die. People, like alcoholics, or people with something like crohn's, these people are walking around with not much phosphorous in them. For me i believe its the aki, the acute kidney injury I received during my lovely time at arca being poisoned with refrigerant. It seems this one specific part of my kidneys are busted, all other function is perfectly normal. If it was long covid, everything would be out of whack, like calcium, magnesium, vitamin d, the only thing that is off for me is the phosphorous and the uric acid, which is handled by the same region in the kidney.
But here is what gets me. They have known this since at least 2021, if not 2020. But whats the standard protocol?
Stick them on a ventilator, and give them something Remdesivir , which will destroy your kidneys if you have any kind of kidney impairment because it cant metabolize it properly.
Remdesivir is not recommended for use in patients with an estimated glomerular filtration rate (eGFR) less than 30 mL/min1. 73 m2 owing to the presence of excipients3 that may accumulate in kidney dysfunction and worsen kidney or hepatic outcomes.
How many people were just straight up murdered by dr. fauci? Wasnt he the one who recommended this procedure?